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Paper Details

GCN5 contributes to intracellular lipid accumulation in human primary cardiac stromal cells from patients affected by Arrhythmogenic cardiomyopathy.
J Cell Mol Med
4
2022
ACM, ACM CStCs, Arrhythmogenic cardiomyopathy, CStCs, CTR, GCN5, MB-3, cardiac diseases, cardiac fibro-fatty replacement, genetic disease, heart failure, histone acetyltransferase, human, human primary cardiac, human primary cardiac stromal cells, oxygen, patients, sudden cardiac death
Author NameAffiliation
Luisa FocoInstitute for Biomedicine, Affiliated Institute of the University of Lubeck
Silvano PiazzaUniversita degli Studi di Trento
Silvano PiazzaInternational Centre for Genetic Engineering and Biotechnology
Silvano PiazzaUniversita degli Studi di Trento
Silvano PiazzaInternational Centre for Genetic Engineering and Biotechnology
Peter P PramstallerInstitute for Biomedicine, Affiliated Institute of the University of Lubeck
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