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Paper Details

Hepatitis B Virus Evasion From Cyclic Guanosine Monophosphate-Adenosine Monophosphate Synthase Sensing in Human Hepatocytes.
Hepatology
65
2018
Adenosine Monophosphate, Chronic hepatitis B virus (HBV) infection, Cyclic, Cyclic Guanosine Monophosphate-Adenosine Monophosphate Synthase, DNA sensor, Guanosine Monophosphate, HBV, HBV-infected human liver chimeric mice, Hepatitis B Virus, Human, Human Hepatocytes, cGAS, cGAS effector gene, cancer, cell culture models, chronic liver disease, cyclic, cyclic guanosine monophosphate-adenosine monophosphate synthase, guanosine monophosphate-adenosine monophosphate, hepatocyte, hepatoma cell lines, human, human liver chimeric, humanized, infectious cell culture model, mice, primary human hepatocytes, relaxed-circular HBV DNA, viral, viral covalently closed circular DNA, viral genome
Author NameAffiliation
Nathalie PochetAnn Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Broad Institute of MIT and Harvard
Nathalie PochetAnn Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Broad Institute of MIT and Harvard
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