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Paper Details

ERBB3 overexpression due to miR-205 inactivation confers sensitivity to FGF, metabolic activation, and liability to ERBB3 targeting in glioblastoma.
Cell Rep
16
2021
AKT, ERBB3, FGF, FGFR, GBM, GBM stem-like cells, GBMs, PI3K, brain tumor, fibroblast growth factor receptor, glioblastoma, human, mTOR, miR, miR-205, oncosuppressor, patient, patient-derived stem-like cells, patients, xenotransplants
Author NameAffiliation
Bianca PolloFondazione IRCCS Istituto Neurologico C. Besta
Gaetano FinocchiaroFondazione IRCCS Istituto Neurologico C. Besta, IRCCS Ospedale San Raffaele
Gaetano FinocchiaroFondazione IRCCS Istituto Neurologico C. Besta, IRCCS Ospedale San Raffaele
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