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Paper Details

Transcription factors operate across disease loci, with EBNA2 implicated in autoimmunity.
Nat Genet
211
2018
EBNA2, Epstein-Barr virus, Epstein-Barr virus EBNA2 protein, GATA3, allele, androgen, androgen receptor, autoimmunity, breast cancer, celiac disease, disease loci, human, inflammatory bowel disease, juvenile idiopathic arthritis, multiple loci, multiple sclerosis, prostate cancer, regulatory regions, rheumatoid arthritis, risk loci, systemic lupus erythematosus, systemic lupus erythematosus risk loci, type 1 diabetes
Author NameAffiliation
John B HarleyCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
John B HarleyCincinnati Children's Hospital Medical Center
John B HarleyCincinnati Children's Hospital Medical Center
John B HarleyUniversity of Cincinnati College of Medicine
John B HarleyUS Department of Veterans Affairs Medical Center
Leah C KottyanCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
Leah C KottyanUniversity of Cincinnati College of Medicine
Matthew T WeirauchCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
Matthew T WeirauchCincinnati Children's Hospital Medical Center
Matthew T WeirauchUniversity of Cincinnati College of Medicine
Matthew T WeirauchCincinnati Children's Hospital Medical Center
Matthew T WeirauchCenter for Autoimmune Genomics and Etiology, Cincinnati Children's Hospital Medical Center
Matthew T WeirauchCincinnati Children's Hospital Medical Center
Matthew T WeirauchUniversity of Cincinnati College of Medicine
Matthew T WeirauchCincinnati Children's Hospital Medical Center
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Release 1.2