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Paper Details

Somatic mutations affect key pathways in lung adenocarcinoma.
Nature
2056
2008
623 genes, APC, ATM, DNA, DNA repair defects, EGFR homologue, EPHA3, ERBB4, KDR, LRP1B, NF1, NTRK genes, PTPRD, RB1, cancer, cancers, ephrin receptor genes, genes, human, lung adenocarcinoma, lung adenocarcinomas, primary, primary lung adenocarcinoma, primary tumours, single nucleotide polymorphism array, tumour, tumour suppressor genes, tyrosine kinases, vascular endothelial growth factor receptor KDR
Author NameAffiliation
Li DingThe Genome Center at Washington University, Washington University School of Medicine
Li DingThe Genome Center at Washington University, Washington University School of Medicine
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