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Paper Details

Vasopressin type 1A receptor deletion enhances cardiac contractility, β-adrenergic receptor sensitivity and acute cardiac injury-induced dysfunction.
Clinical Science
5
2016
-adrenergic receptor, AR, HF, PLB, V1AR, Vasopressin, Vasopressin type 1A receptor, adrenergic receptor, cAMP, cTnI, cardiac dysfunction, cardiac injury, cardiac troponin I, cardiomyocyte, heart failure, human, ischaemic injury, mice, myocyte, myocytes, phospholamban
Author NameAffiliation
Walter J KochCenter for Translational Medicine, Lewis Katz School of Medicine, Temple University
Joseph Y CheungCenter for Translational Medicine, Lewis Katz School of Medicine, Temple University
Joseph Y CheungLewis Katz School of Medicine, Temple University
Arthur M FeldmanLewis Katz School of Medicine, Temple University
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Datasets

Release 1.2