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Paper Details

Cytoplasmic chromatin triggers inflammation in senescence and cancer.
Nature
753
2017
Chromatin, Cytoplasmic chromatin, Mice, STING, cGAS, cancer, cancer cells, cancers, chromatin, chronic inflammation, cyclic GMP-AMP, cyclic GMP-AMP synthase, cytoplasm, cytoplasmic, cytoplasmic chromatin, cytoplasmic chromatin fragments, deficient in STING, genomic DNA, human, inflammation, inflammation-related disorders, mice, oncogenes, oncogenic RAS, primary cells, primary human cells, pro-inflammatory gene, senescence, stimulator of interferon genes
Author NameAffiliation
Klaus H KaestnerUniversity of Pennsylvania
Klaus H KaestnerCenter for Molecular Studies in Digestive and Liver Diseases, University of Pennsylvania
Benjamin A GarciaPerelman School of Medicine, University of Pennsylvania
Benjamin A GarciaPenn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania
Benjamin A GarciaPerelman School of Medicine, University of Pennsylvania
Benjamin A GarciaPerelman School of Medicine, University of Pennsylvania
Benjamin A GarciaPerelman School of Medicine, University of Pennsylvania
Benjamin A GarciaPenn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania
Peter D AdamsBeatson Institute for Cancer Research
Peter D AdamsInstitute of Cancer Sciences, College of Medical, University of Glasgow
Peter D AdamsSanford Burnham Prebys Medical Discovery Institute
Shelley L BergerPerelman School of Medicine, University of Pennsylvania
Shelley L BergerUniversity of Pennsylvania
Shelley L BergerPenn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania
Shelley L BergerUniversity of Pennsylvania
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