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Paper Details

The impact of increasing levels of blood C-reactive protein on the inflammatory loci SPI1 and CD33 in Alzheimer's disease.
Transl Psychiatry
5
2022
AD, AD-associated genes, APOE, APOE 4, APOE 4 allele, Alzheimer's Disease, Alzheimer's disease, Apolipoprotein 4, CD33, CD33 genotype, CD33 rs3865444, CLU, CLU rs9331896-, CRP, MCI, Mild Cognitive Impairment, SNPs, SPI1, SPI1 genotype, SPI1 rs1057233, SPI1 rs1057233-, blood, blood C-reactive protein, blood CRP, genetic variants, inflammatory loci, p-Tau, participants, patients, people, rs3865444, serum CRP, single nucleotide polymorphisms, t-Tau
Author NameAffiliation
John J FarrellBoston University School of Medicine
Kathryn L LunettaBoston University School of Public Health
Kathryn L LunettaBoston University School of Medicine
Rhoda AuBoston University School of Medicine
Rhoda AuBoston University School of Public Health
Rhoda AuAlzheimer's Disease Research Center, Boston University School of Medicine
Rhoda AuBoston University School of Medicine
Rhoda AuBoston University School of Medicine
Lindsay A FarrerBoston University School of Medicine
Lindsay A FarrerBoston University School of Public Health
Lindsay A FarrerAlzheimer's Disease Research Center, Boston University School of Medicine
Lindsay A FarrerBoston University School of Public Health
Lindsay A FarrerBoston University School of Medicine
Lindsay A FarrerBoston University School of Medicine
Lindsay A FarrerBoston University School of Medicine
Xiaoling ZhangBoston University School of Medicine
Xiaoling ZhangBoston University School of Public Health
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