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Paper Details

Differential Kinase Activity Across Prostate Tumor Compartments Defines Sensitivity to Target Inhibition.
Cancer Res
3
2022
CTC, Cancer, EpCAM, EpCAM-high cells, EpCAM-high epithelial cells, PI3K, PTEN, PTEN-deleted orthotopic prostate cancer xenograft models, Prostate, Prostate Tumor, RTK, bone metastases, c-MET, c-MET protein, circulating tumor cells, drug, drug-resistant EpCAM-low populations, mTOR, mesenchymal CTC populations, metastatic, metastatic prostate and breast cancer, patient, patients, poorly proliferating EpCAM-low mesenchymal populations, primary tumors, prostate cancer, receptor tyrosine kinases, tumor, tyrosine
Author NameAffiliation
Wilhelm HaasMassachusetts General Hospital Center for Cancer Research, Harvard Medical School
David T MiyamotoMassachusetts General Hospital Center for Cancer Research, Harvard Medical School
Mehmet TonerCenter for Engineering in Medicine and Surgery, Massachusetts General Hospital, Harvard Medical School
Mehmet TonerShriners Hospital for Children
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Release 1.2