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Paper Details

Microenvironmental CXCL12 deletion enhances Flt3-ITD acute myeloid leukemia stem cell response to therapy by reducing p38 MAPK signaling.
Leukemia
6
2023
-abundant reticular (CAR) cells, AML, AML LSC, AML cells, CXCL12, CXCR4, FLT3, FLT3-ITD, FLT3-ITD AML LSC, FLT3-ITD LSC, Flt3, Flt3-, Flt3-ITD, Flt3-ITD AML, Flt3-ITD AML LSC, Flt3-ITD acute myeloid leukemia stem cell, Flt3-internal tandem duplication, Fms-like tyrosine kinase 3, ITD, LSC, MSC, Tet2, acute myeloid leukemia, human, human Flt3-ITD, leukemia, leukemia stem cells, mice, mouse, murine, murine and human Flt3-ITD AML LSC, p38, p38 MAPK, p38 mitogen-activated protein kinase, patients, primitive ST-HSC population, tyrosine
Author NameAffiliation
David K CrossmanUniversity of Alabama at Birmingham
David K CrossmanUniversity of Alabama at Birmingham
Robert S WelnerUniversity of Alabama at Birmingham
Robert S WelnerUniversity of Alabama at Birmingham
Ravi BhatiaUniversity of Alabama at Birmingham
Ravi BhatiaUniversity of Alabama at Birmingham
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Release 1.2