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Paper Details

Sexual dimorphism in atrophic effects of topical glucocorticoids is driven by differential regulation of atrophogene REDD1 in male and female skin.
Oncotarget
7
2020
ER, GR, REDD1, REDD1 mRNA, atrophogene REDD1, ear edema, estradiol, estrogen, estrogen receptor, estrogen receptor (ER) target gene, estrogen response elements, fluocinolone acetonide, human, human keratinocytes, mice, patients, promoter, regulated in development and DNA damage 1, skin atrophy, steroid atrophy
Author NameAffiliation
Ben ReadheadIcahn School of Medicine at Mount Sinai
Ben ReadheadCurrent address: ASU-Banner Neurodegenerative Disease Research Center, Arizona State University
Joel T DudleyIcahn School of Medicine at Mount Sinai
Joel T DudleyIcahn School of Medicine at Mount Sinai
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Release 1.2