Skip to Main Content

Paper Details

Human airway mast cells proliferate and acquire distinct inflammation-driven phenotypes during type 2 inflammation.
Sci Immunol
76
2021
CD38highCD117high, CD38highCD117high MCs, CRSwNP, CRSwNP MCT, Human, Human airway mast cells, MC, MC hyperplasia, MC subsets, MCT, MCTC, MCTC subsets, MCs, Mast cells, T2, T2 cytokine-mediated inflammatory disease, T2 disease, T2 inflammation, aspirin, aspirin-exacerbated respiratory disease, asthma, chronic rhinosinusitis, chymase, circulating MC progenitors, epithelial MCs, fibrotic diseases, human, nasal polyp MCs, nasal polyposis, nasal polyps, patients, subepithelial MCs, tryptase, type 2 (T2) allergic inflammatory diseases of the airway, type 2 inflammation
Author NameAffiliation
Jose Ordovas-MontanesBoston Children's Hospital
Jose Ordovas-MontanesHarvard Stem Cell Institute
Jose Ordovas-MontanesBroad Institute of MIT and Harvard
Jose Ordovas-MontanesHarvard Medical School
Bonnie Berger
Alex K ShalekBroad Institute of MIT and Harvard
Alex K ShalekInstitute for Medical Engineering and Science (IMES), and Koch Institute for Integrative Cancer Research
Alex K ShalekRagon Institute of MGH
Alex K ShalekBroad Institute of MIT and Harvard
Alex K ShalekInstitute for Medical Engineering and Science (IMES), and Koch Institute for Integrative Cancer Research
Alex K ShalekRagon Institute of MGH
  • 1 - 11

Datasets

Release 1.2