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Paper Details

Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt<sup>Q111/+</sup> model of Huntington's disease.
Sci Rep
14
2017
B6, HD, HttQ111, HttQ111/+, Huntington's disease, NIIs, anxiety, depression, glia, gliosis, mice, motor impairment, mouse, neurodegeneration, striatal, synapse
Author NameAffiliation
Seth A AmentInstitute for Systems Biology
Seth A AmentInstitute for Genome Sciences and Department of Psychiatry, University of Maryland School of Medicine
Seth A AmentInstitute for Systems Biology
Seth A AmentInstitute for Genome Sciences and Department of Psychiatry, University of Maryland School of Medicine
Cory C FunkInstitute for Systems Biology
Marcy E MacDonaldCenter for Human Genetic Research, Massachusetts General Hospital, Harvard Medical School
Nathan D PriceInstitute for Systems Biology
Nathan D PriceInstitute for Systems Biology
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