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Paper Details

DACH1 protects podocytes from experimental diabetic injury and modulates PTIP-H3K4Me3 activity.
J Clin Invest
23
2021
DACH1, DKD, Dach1, Dachshund, Dachshund homolog 1, H3K4Me3, PTIP, Pax transactivation-domain interacting protein, Podocyte, Podocyte-specific, Ptip, RNA, binding sites, diabetes, diabetic injury, diabetic kidney disease, histone H3 lysine 4 trimethylation, hyperglycemia, injury-resistant podocytes, injury-susceptible podocytes, lysine, mice, patients, podocyte, podocytes, promoter Dach1-binding sites, promoter-, renal hypoplasia, target gene, target genes, upregulated genes
Author NameAffiliation
Song JiangNational Clinical Research Center of Kidney Diseases, Jinling Hospital, Nanjing University School of Medicine
Evren U AzelogluIcahn School of Medicine at Mount Sinai
Weijia ZhangIcahn School of Medicine at Mount Sinai
Zhihong LiuNational Clinical Research Center of Kidney Diseases, Jinling Hospital, Nanjing University School of Medicine
John Cijiang HeIcahn School of Medicine at Mount Sinai
Vivette D D'AgatiColumbia University Medical Center
Vivette D D'AgatiColumbia University Medical Center
Katalin SusztakPerelman School of Medicine at University of Pennsylvania
Lewis KaufmanIcahn School of Medicine at Mount Sinai
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