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Paper Details

Single-nucleus chromatin accessibility profiling highlights regulatory mechanisms of coronary artery disease risk.
Nat Genet
45
2022
10q23, 3q22, CAD, Coronary artery disease, LIPA, MRAS, PRDM16, TBX2, accessible sites, cell types, cell-type-specific elements, cellular clusters, chromatin, cis, coronary artery disease, elements, fibromyocytes, functional variants, inflammatory disease, noncoding DNA sequences, patients, regulatory elements, transposase
Author NameAffiliation
Shengen Shawn HuCenter for Public Health Genomics, University of Virginia
Anshul KundajeStanford University School of Medicine
Anshul KundajeStanford University
Anshul KundajeStanford University School of Medicine
Anshul KundajeStanford University
Suna Onengut-GumuscuCenter for Public Health Genomics, University of Virginia
Suna Onengut-GumuscuUniversity of Virginia
Suna Onengut-GumuscuUniversity of Virginia
Euan A AshleyStanford University School of Medicine
Euan A AshleyStanford University
Thomas QuertermousStanford University
Aloke V FinnCVPath Institute
Jason C KovacicCardiovascular Research Institute, Icahn School of Medicine at Mount Sinai
Jason C KovacicSt. Vincent's Clinical School, University of New South Wales
Jason C KovacicVictor Chang Cardiac Research Institute
Chongzhi ZangCenter for Public Health Genomics, University of Virginia
Chongzhi ZangUniversity of Virginia
Chongzhi ZangUniversity of Virginia
Clint L MillerCenter for Public Health Genomics, University of Virginia
Clint L MillerUniversity of Virginia
Clint L MillerRobert M. Berne Cardiovascular Research Center, University of Virginia
Clint L MillerUniversity of Virginia
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