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Paper Details

CRISPR editing of CCR5 and HIV-1 facilitates viral elimination in antiretroviral drug-suppressed virus-infected humanized mice.
2
2023
C-C chemokine receptor type five, CCR5, CD34, CD4, CRISPR, Cas9, HIV, HIV-1, HIV-1 DNA fragments, HIV-1-infected CD34+ NSG-humanized mice, abacavir, cabotegravir, ester, human, human CD4 T cell, humanized, infected human cells, lamivudine, mice, proviral DNA, rilpivirine
Author NameAffiliation
Prasanta K DashCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Rafal KaminskiCenter for Neurovirology and Gene Editing, Lewis Katz School of Medicine at Temple University
Hang SuCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Pietro MancusoCenter for Neurovirology and Gene Editing, Lewis Katz School of Medicine at Temple University
Brady SillmanCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Saumi MathewsCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Rahsan SariyerCenter for Neurovirology and Gene Editing, Lewis Katz School of Medicine at Temple University
Rodney Lee MosleyCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Larisa Y PoluektovaCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Shohreh AminiCenter for Neurovirology and Gene Editing, Lewis Katz School of Medicine at Temple University
Shohreh AminiCollege of Science and Technology, Temple University
Santhi GorantlaCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Tricia H BurdoCenter for Neurovirology and Gene Editing, Lewis Katz School of Medicine at Temple University
Benson EdagwaCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Howard E GendelmanCenter for Neurodegenerative Diseases, University of Nebraska Medical Center
Kamel KhaliliCenter for Neurovirology and Gene Editing, Lewis Katz School of Medicine at Temple University
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Release 1.2