Skip to Main Content

Paper Details

Clonally expanding smooth muscle cells promote atherosclerosis by escaping efferocytosis and activating the complement cascade.
Proc Natl Acad Sci U S A
73
2020
Atherosclerosis, CD47, SMC, SMCs, atherosclerosis, atherosclerotic plaques, cardiovascular disease, cholesterol, clonally expanding SMC, don, heart attack, humans, hyperproliferative cell, inflamed phagocytes, inflammatory cells, macrophages, mature SMC, smooth muscle cells, stroke, vascular smooth muscle cells
Author NameAffiliation
Abhiram S RaoStanford Cardiovascular Institute, Stanford University
Abhiram S RaoStanford University
James R PriestStanford University School of Medicine
Clint L MillerCenter for Public Health Genomics, University of Virginia
Clint L MillerUniversity of Virginia
Clint L MillerUniversity of Virginia
Lars LindUppsala University
Simon KoplevIcahn Institute for Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Simon KoplevIcahn Institute for Genomics and Multiscale Biology, Icahn School of Medicine at Mount Sinai
Erik IngelssonStanford Cardiovascular Institute, Stanford University
Erik IngelssonUppsala University
Erik IngelssonStanford University School of Medicine
Erik IngelssonStanford Cardiovascular Institute, Stanford University
Erik IngelssonUppsala University
Erik IngelssonStanford University School of Medicine
  • 1 - 15

Datasets

Release 1.2