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Paper Details

Suppression of Myeloid Cell Arginase Activity leads to Therapeutic Response in a NSCLC Mouse Model by Activating Anti-Tumor Immunity.
J Immunother Cancer
92
2019
ARG1/2, CD3, CD66b, GEMM, KRASG12D, KRASG12D GEMM, KrasG12D, MDSC, Mouse, Myeloid Cell, Myeloid Cell Arginase, NOS, NSCLC, RNA, T, T cell, Tumor, amino acid, arginase, arginase 1, arginase 1,2, arginine, autochthonous immunocompetent, autochthonous immunocompetent KrasG12D GEMM, granulocytic myeloid cells, human, lung adenocarcinoma, lung adenocarcinomas, lung cancer, lung tumor, lung tumors, lymphocyte, mouse, mouse lung adenocarcinomas, murine, murine and human non-small cell (NSCLC) lung cancer lines, myeloid, myeloid cells, myeloid derived suppressor cell, nitric oxide, nitric oxide synthase, non-small cell (NSCLC) lung cancer, patient, sorted cell populations, sorted myeloid cells, splenocyte, squamous tumors, tumor, tumors
Author NameAffiliation
Venkat S MalladiUniversity of Texas Southwestern Medical Center
Venkat S MalladiUniversity of Texas Southwestern Medical Center
Peter S HammermanDana Farber Cancer Institute
Peter S HammermanBroad Institute of Harvard and MIT
Peter S HammermanNovartis Institutes for Biomedical Research
Peter S HammermanDana Farber Cancer Institute
Peter S HammermanNovartis Institutes for Biomedical Research
Peter S HammermanBroad Institute of Harvard and MIT
Chad V PecotUniversity of North Carolina Chapel Hill, Lineberger Cancer Center
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