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Paper Details

Mutant P53 induces MELK expression by release of wild-type P53-dependent suppression of FOXM1.
NPJ Breast Cancer
14
2020
-, -binding site, -null cells, E2F1, E2F1A, ER, ER-positive breast cancers, FOXM1, FOXM1 promoter, FOXM1-binding site, MELK, MELK mRNA, MELK promoter, MELK protein, Mutant P53, TNBC, TNBCs, Triple-negative breast cancer, breast cancer, leucine, maternal embryonic leucine zipper kinase, p53, p53 consensus response elements, p53 gene, p53-mutant, p53-mutant TNBC cells, p53-mutant breast cancers, p53-mutant vs. p53 wild-type breast cancer cells, p53-null cells, responsive region, type p53, wild-type P53, wild-type p53, wild-type p53 target genes
Author NameAffiliation
Gordon B MillsThe University of Texas MD Anderson Cancer Center
Gordon B MillsOHSU Knight Cancer Institute, Oregon Health and Science University
Gordon B MillsThe University of Texas MD Anderson Cancer Center
Gordon B MillsOHSU Knight Cancer Institute, Oregon Health and Science University
Powel H BrownThe University of Texas MD Anderson Cancer Center
Powel H BrownBaylor College of Medicine
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