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Paper Details

Quantitative proteomics identify Tenascin-C as a promoter of lung cancer progression and contributor to a signature prognostic of patient survival.
Proc Natl Acad Sci U S A
101
2017
Fibronectin, S100 proteins, S100A10, S100A11, TNC, Tenascin-C, Tnc, adenocarcinoma, cancer, endogenous gene, fibrosis, fibrotic, fibrotic lung, human, lung, lung adenocarcinoma, lung adenocarcinoma cells, lung cancer, lung fibrosis, lung tumors, lymph node metastases, metastases, mouse, normal tissue, patient, patients, primary lung tumors, primary tumors, three-gene expression signature, transcription factor Nkx2-1, tumor
Author NameAffiliation
Alexandra NabaDavid H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology
Alexandra NabaMassachusetts Institute of Technology
Alexandra NabaDavid H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology
Alexandra NabaMassachusetts Institute of Technology
Caroline C KimDavid H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology
Caroline C KimMassachusetts Institute of Technology
Monte M WinslowDavid H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology
Monte M WinslowMassachusetts Institute of Technology
Richard O HynesDavid H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology
Richard O HynesMassachusetts Institute of Technology
Richard O HynesHoward Hughes Medical Institute, Massachusetts Institute of Technology
Tyler JacksDavid H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology
Tyler JacksMassachusetts Institute of Technology
Tyler JacksHoward Hughes Medical Institute, Massachusetts Institute of Technology
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