Skip to Main Content

Paper Details

A robust error model for iTRAQ quantification reveals divergent signaling between oncogenic FLT3 mutants in acute myeloid leukemia.
Mol Cell Proteomics
69
2010
D835Y, FLT3, FLT3 mutants, FLT3 receptor tyrosine kinase, FLT3-D835Y, SHP1, acute myeloid leukemia, downstream targets, leukemogenesis, oncogenic FLT3 mutants, phosphorylation sites, tyrosine, tyrosine phosphatase

Datasets