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Paper Details

<i>HLA</i> and autoantibodies define scleroderma subtypes and risk in African and European Americans and suggest a role for molecular mimicry.
Proc Natl Acad Sci U S A
45
2020
Author NameAffiliation
Maureen D MayesDivision of Rheumatology and Clinical Immunogenetics, University of Texas McGovern Medical School
Ayo P DoumateyCenter for Research on Genomics and Global Health, National Human Genome Research Institute, National Institutes of Health
Daniel ShrinerCenter for Research on Genomics and Global Health, National Human Genome Research Institute, National Institutes of Health
Paula S RamosMedical University of South Carolina
Lindsey A CriswellRosalind Russell/Ephraim P. Engleman Rheumatology Research Center, University of California san francisco
S Louis BridgesDivision of Clinical Immunology and Rheumatology, University of Alabama at Birmingham
S Louis BridgesDivision of Clinical Immunology and Rheumatology, University of Alabama at Birmingham
Benjamin D KormanUniversity of Rochester Medical Center
Settara C ChandrasekharappaNational Human Genome Research Institute, National Institutes of Health
James C MullikinNational Human Genome Research Institute
James C MullikinNational Human Genome Research Institute
Adebowale AdeyemoCenter for Research on Genomics and Global Health, National Human Genome Research Institute, National Institutes of Health
Adebowale AdeyemoCenter for Research on Genomics and Global Health, National Human Genome Research Institute, National Institutes of Health
Charles N RotimiCenter for Research on Genomics and Global Health, National Human Genome Research Institute, National Institutes of Health
Charles N RotimiCenter for Research on Genomics and Global Health, National Human Genome Research Institute, National Institutes of Health
Daniel L KastnerNational Human Genome Research Institute, National Institutes of Health
Elaine F RemmersNational Human Genome Research Institute, National Institutes of Health
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