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Paper Details

Inhibition of autophagy at a late stage enhances imatinib-induced cytotoxicity in human malignant glioma cells.
Int J Cancer
147
2009
3-methyladenine, Atg5, Imatinib, Imatinib mesylate, Malignant gliomas, RTA 203, U373-MG cells, U87-MG, U87-MG and U373-MG cells, autophagy, bafilomycin A1, human, human malignant glioma cells, imatinib, leukemias, malignant glioma, patients, primary tumors of the central nervous system, small interfering RNA, tumor, tumor cells, tumors, tyrosine, tyrosine kinase
Adenine, Antineoplastic Agents, Autophagy, Autophagy-Related Protein 5, Benzamides, Brain Neoplasms, Bridged Bicyclo Compounds, Heterocyclic, Cell Line, Tumor, Cisplatin, Extracellular Signal-Regulated MAP Kinases, Glioma, Humans, Imatinib Mesylate, MAP Kinase Signaling System, Macrolides, Membrane Potential, Mitochondrial, Microtubule-Associated Proteins, Paclitaxel, Piperazines, Pyrimidines
Author NameAffiliation
Takashi ShinguThe University of Texas M. D. Anderson Cancer Center
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