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Paper Details

A P53-Independent DNA Damage Response Suppresses Oncogenic Proliferation and Genome Instability.
Cell Rep
23
2020
ATR, DNA double-strand break, Mre11, Mre11 complex, Nbs1, P53, PARP, Rad3 related, Rad50, ataxia telangiectasia, human, hypomorphic Mre11 allele, p53, p53-deficient breast cancers, poly (ADP-ribose), poly (ADP-ribose) polymerase, primary mammary epithelial cell, primary mammary epithelial cell model, triple-negative breast cancers, tumor
Author NameAffiliation
Luciano G MartelottoMemorial Sloan Kettering Cancer Center
Lisle E MoseLineberger Comprehensive Cancer Center, University of North Carolina at Chapel Hill
Lisle E MoseLineberger Comprehensive Cancer Center, University of North Carolina at Chapel Hill
Simon N PowellMemorial Sloan Kettering Cancer Center
Hannah Y WenMemorial Sloan Kettering Cancer Center
Jorge S Reis-FilhoMemorial Sloan Kettering Cancer Center
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