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Paper Details

The <i>CIC-ERF</i> co-deletion underlies fusion-independent activation of ETS family member, ETV1, to drive prostate cancer progression.
Elife
4
2022
-ERF, <, <i>, <i>CIC</i>, <i>CIC</i> and <i>ERF</i>-deficient prostate cancer, <i>ERF, <i>ERF</i>, CIC, Capicua, ERF, ETS, ETS factor, ETS family, ETS gene, ETS transcription factor, ETS2 repressor factor, ETV1, Human, chromosome 19q13, human, prostate cancer, prostate tumors, proximal regulatory element, tumor
Author NameAffiliation
Eric J SmallUniversity of California san francisco
Eric J SmallHelen Diller Family Comprehensive Cancer Center, University of California san francisco
Felix Y FengUniversity of California san francisco
Felix Y FengHelen Diller Family Comprehensive Cancer Center, University of California san francisco
Felix Y FengUniversity of California san francisco
Franklin W HuangUniversity of California san francisco
Franklin W HuangHelen Diller Family Comprehensive Cancer Center, University of California san francisco
Franklin W HuangUniversity of California san francisco
Franklin W HuangHelen Diller Family Comprehensive Cancer Center, University of California san francisco
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