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Paper Details

Identification of a novel TP53 cancer susceptibility mutation through whole-genome sequencing of a patient with therapy-related AML.
JAMA
126
2011
3-kilobase deletion, BRCA1, BRCA2, RNA, Skin (normal), TP53, TP53 cancer susceptibility mutation, bone marrow (leukemia) DNA, cancer, cancer genome, cancer susceptibility genes, coding genes, exons 7-9, indels, inherited cancer, leukemia, leukemia DNA, leukemia genome, normal skin DNA, paired, patient, patients, single, single-nucleotide polymorphism, t-AML, therapy-related AML, therapy-related acute myeloid leukemia, tumors, uniparental disomy, whole
Author NameAffiliation
Daniel C LinkSiteman Cancer Center, Washington University
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Datasets

Release 1.2