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Paper Details

Dnmt3a deletion cooperates with the Flt3/ITD mutation to drive leukemogenesis in a murine model.
Oncotarget
14
2016
/, /ITD, AML, Acute Myeloid Leukemia, DNMT3A, DNMT3A mutations, Dnmt3a, Dnmt3af/+, Dnmt3af/f, FLT3, FLT3 and, FLT3/ITD, Flt3, Flt3ITD/+, ITD, LT-HSC, LT-HSCs, MPN, T, T-ALL, acute leukemia, allele, endogenous Flt3 locus, hematopoietic, hematopoietic neoplasms, leukemogenesis, mice, mouse, mutant DNMT3A, myeloproliferative neoplasm, neoplasms
Author NameAffiliation
Li LiJohns Hopkins University School of Medicine
Li LiJohns Hopkins University School of Medicine
Donald SmallJohns Hopkins University School of Medicine
Donald SmallJohns Hopkins University School of Medicine
Donald SmallJohns Hopkins University School of Medicine
Donald SmallJohns Hopkins University School of Medicine
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Release 1.2