Skip to Main Content

Paper Details

Loss of ARID1A Promotes Epithelial-Mesenchymal Transition and Sensitizes Pancreatic Tumors to Proteotoxic Stress.
Cancer Res
23
2021
ARID1A, ARID1A defects, ARID1A-deficient cells, AT-rich interaction domain-containing protein 1A, AUY922, HSP90, NVP, PDAC, PDAC cells, PDAC tumors, Pancreatic Tumors, SNF, SWI, SWI/SNF complex genes, SWItch, Sucrose, Sucrose non-fermentable, anticancer, cancer, cancers, human, mesenchymal phenotype, pancreatic ductal adenocarcinomas, patients, quasi-mesenchymal phenotype, tumor, tumors
Author NameAffiliation
Justin K HuangThe University of Texas MD Anderson Cancer Center
Yonathan Lissanu DeribeThe University of Texas MD Anderson Cancer Center
Anirban MaitraThe University of Texas MD Anderson Cancer Center
Anirban MaitraSheikh Ahmed Bin Zayed Al Nahyan Center for Pancreatic Cancer Research, The University of Texas MD Anderson Cancer Center
Pavlos MsaouelThe University of Texas MD Anderson Cancer Center
Andrea VialeThe University of Texas MD Anderson Cancer Center
Timothy P HeffernanThe University of Texas MD Anderson Cancer Center
Christopher A BristowThe University of Texas MD Anderson Cancer Center
Christopher A BristowThe University of Texas MD Anderson Cancer Center
  • 1 - 9

Datasets

Release 1.2