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Paper Details

Mitochondrial DNA alterations underlie an irreversible shift to aerobic glycolysis in fumarate hydratase-deficient renal cancer.
Sci Signal
48
2021
HLRCC, HLRCC tumors, Hereditary leiomyomatosis and renal cell carcinoma, Krebs, Mitochondrial DNA, cancer, fumarate, fumarate hydratase, fumarate hydratase-deficient renal cancer, glucose, glucose-dependent human cancer, human, mitochondrial DNA, mtDNA, patients, tumors
Author NameAffiliation
Daniel R CrooksCenter for Cancer Research, National Cancer Institute
Christopher J RickettsCenter for Cancer Research, National Cancer Institute
Ruth M PfeifferNational Cancer Institute
Ruth M PfeifferNational Cancer Institute
Thomas RiedCenter for Cancer Research, National Cancer Institute
Thomas RiedCenter for Cancer Research, National Cancer Institute
Darawalee WangsaCenter for Cancer Research, National Cancer Institute
Darawalee WangsaCenter for Cancer Research, National Cancer Institute
Maria J MerinoNational Cancer Institute
Maria J MerinoNational Cancer Institute
Tracey A RouaultEunice Kennedy Shriver National Institute of Child Health and Human Development
William Marston LinehanCenter for Cancer Research, National Cancer Institute
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