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Paper Details

Pathogenic monoallelic variants in GLIS3 increase type 2 diabetes risk and identify a subgroup of patients sensitive to sulfonylureas.
0
2023
Diabetes, GLIS, GLIS family, GLIS3, GLIS3 protein, GLIS3 variants, INS promoter, Obesity, P, P/LP GLIS3 variants, P/LP variants, P/LP) variants, beta cell, cholesterol, exons, insulin, last two exons, luciferase, mouse, pancreatic beta cell, participants, patients, sulfonylureas, susceptibility genes, syndromic neonatal diabetes, type 2 diabetes, zinc
Author NameAffiliation
Beverley BalkauInserm U8 Clinical Epidemiology, Center for Research in Epidemiology and Population Health, Paris-Saclay University, Paris-Sud University
Guillaume Charpentier
Michel MarreInstitut Necker-Enfants Malades, Inserm, Universite de Paris
Michel Marre
Philippe FroguelInserm UMR3, CNRS UMR8199, European Genomic Institute for Diabetes (EGID), Universite de Lille, Institut Pasteur de Lille, Lille University Hospital
Philippe FroguelImperial College London
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