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Paper Details

Hyperglycemia Acutely Increases Cytosolic Reactive Oxygen Species via -linked GlcNAcylation and CaMKII Activation in Mouse Ventricular Myocytes.
Circulation Research
68
2020
(R)--[[(1,2-dihydro-2-oxo-6-quinolinyl)sulfonyl], 2',7'-dichlorodihydrofluorescein diacetate, Ca2+, Ca2+/calmodulin-dependent kinase II, CaMKII, DCF, Diabetes mellitus, Diabetic hyperglycemia, GKT137831, GlcNAc, Glu, Gp91ds-tat peptide, Grx, Hi, Hi-Glu, Hyperglycemia, KN-93, L-NAME, L-NIO, L-NPA, Mouse, Mouse Ventricular Myocytes, N-acetylglucosamine, NADPH, NO, NO synthase, NOS, NOX (NADPH oxidase) 2, NOX2, NOX4, O, O-GlcNAc, Oxygen, S280A, Thiamet G, ThmG, arrhythmia, arrhythmogenic, cardiac, cardiac myocyte, cardiomyocytes, diabetes mellitus, diabetic hyperglycemia, glucose, mice, mouse, multisystem disease, myocyte, myocytes, oxygen, postinfarction, roGFP2, ventricular myocyte, ventricular myocytes
Author NameAffiliation
Mark MercolaStanford Cardiovascular Institute and Department of Medicine, Stanford University
Joan Heller BrownUniversity of California San Diego
Donald M BersUniversity of California, Davis School of Medicine (S.L.
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