Skip to Main Content

Paper Details

FGFR-inhibitor-mediated dismissal of SWI/SNF complexes from YAP-dependent enhancers induces adaptive therapeutic resistance.
Nat Cell Biol
18
2021
BRG1, FGFR, SNF complexes, SWI, TNBC, TNBC patient-derived xenograft models, YAP, YAP-, YAP-associated enhancers, YAP-dependent enhancers, amino acid, amino acids, cancer, cancer cells, chromatin, fibroblast growth factor receptor, mTORC1, oncogenic, patient, triple-negative breast cancer
Author NameAffiliation
Alok K TewariDana-Farber Cancer Institute, Harvard Medical School
Alok K TewariCenter for Functional Cancer Epigenetics, Dana-Farber Cancer Institute
Xiaole Shirley LiuCenter for Functional Cancer Epigenetics, Dana-Farber Cancer Institute
Xiaole Shirley LiuDana-Farber Cancer Institute, Harvard T.H. Chan School of Public Health
Alex Tokerand Cancer Center, Beth Israel Deaconess Medical Center, Harvard Medical School
Alex TokerHarvard Medical School
Myles BrownDana-Farber Cancer Institute, Harvard Medical School
Myles BrownCenter for Functional Cancer Epigenetics, Dana-Farber Cancer Institute
Myles BrownHarvard Medical School
  • 1 - 9

Datasets

Release 1.2