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Paper Details

<i>RSPO2</i> gene rearrangement: a powerful driver of β-catenin activation in liver tumours.
Gut
24
2019
-, -catenin, -target genes, 46, 8q23, CTNNB1, CTNNB1 gene, CTNNB1 mutation, CTNNB1wild, HCA, HCA subtypes, HCAs, HCC, HCCs, Integration Site Family, MMTV, MMTV Integration Site Family, RSPO2, RSPO2 gene, RSPO2 protein, TERT promoter, WNT, Wingless-Type, b-HCA, b-HCAs, catenin, chromosome, chronic liver disease, hepatocellular carcinoma, human, human telomerase reverse transcriptase promoter, liver tumours, roof plate-specific spondin 2, roof plate-specific spondin 2 (RSPO2) gene, three, three -catenin, three -catenin-activated HCCs
Author NameAffiliation
Thomas LongerichInstitute of Pathology, UniversitatsKlinikum Heidelberg
Thomas LongerichLiver Cancer Center Heidelberg, UniversitatsKlinikum Heidelberg
Volker EndrisInstitute of Pathology, UniversitatsKlinikum Heidelberg
Olaf NeumannInstitute of Pathology, UniversitatsKlinikum Heidelberg
Martina KirchnerInstitute of Pathology, UniversitatsKlinikum Heidelberg
Sebastian UhrigRuprecht Karls University Heidelberg
Sebastian UhrigGerman Cancer Research Center (DKFZ)
Benedikt BrorsGerman Cancer Research Center (DKFZ)
Benedikt BrorsGerman Cancer Research Center (DKFZ)
Jan BudcziesInstitute of Pathology, UniversitatsKlinikum Heidelberg
Peter SchirmacherInstitute of Pathology, UniversitatsKlinikum Heidelberg
Peter SchirmacherLiver Cancer Center Heidelberg, UniversitatsKlinikum Heidelberg
Albrecht StenzingerInstitute of Pathology, UniversitatsKlinikum Heidelberg
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