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Paper Details

Common molecular features of H3K27M DMGs and PFA ependymomas map to hindbrain developmental pathways.
Acta Neuropathol Commun
11
2023
1q, ACVR1, Activin A Receptor Type 1, DMGs, EZH inhibitory protein, EZHIP, H3, H3K27, H3K27 acetylation (H3K27ac)-marked enhancers, H3K27M, H3K27M DMGs, H3K27M mutations, H3K27ac, H3K27me3, H3K27me3-marked genes, PFA, PFA ependymomas, PFAs, PRC2, astrocytic, astrocytic-like tumor cells, cell populations, chromatin, diffuse midline gliomas, ependymomas, group, histone, histone 3, human, lysine, methionine, mutant H3K27M, polycomb repressive complex 2, posterior fossa ependymomas, progenitor populations, scRNA, single, tumor, tumor cells, tumors
Author NameAffiliation
Matthew PunUniversity of Michigan, 1150 W. Medical Center ann arbor
Matthew PunUniversity of Michigan Medical School ann arbor
Matthew PunUniversity of Michigan Medical School ann arbor
Matthew PunUniversity of Michigan ann arbor
Drew PrattCenter for Cancer Research, National Cancer Institute, National Institutes of Health
Bernhard Englinger
Bernhard EnglingerCenter for Cancer Research and Comprehensive Cancer Center, Medical University Vienna
Bernhard EnglingerComprehensive Cancer Center, Medical University of Vienna
Bernhard EnglingerBroad Institute of Harvard and MIT
Arvind RaoUniversity of Michigan Medical School ann arbor
Arvind RaoRogel Cancer Center, University of Michigan Medical School ann arbor
Arvind RaoUniversity of Michigan Medical School ann arbor
Arvind RaoUniversity of Michigan ann arbor
Arvind RaoUniversity of Michigan Medical School ann arbor
Arvind RaoUniversity of Michigan ann arbor
Arvind RaoUniversity of Michigan Medical School ann arbor
Arvind RaoRogel Cancer Center, University of Michigan Medical School ann arbor
Marcin CieslikMichigan Center for Translational Pathology, University of Michigan Medical School ann arbor
Marcin CieslikRogel Cancer Center, University of Michigan Medical School ann arbor
Arul M ChinnaiyanMichigan Center for Translational Pathology, University of Michigan Medical School ann arbor
Arul M ChinnaiyanHoward Hughes Medical Institute, University of Michigan Medical School ann arbor
Arul M ChinnaiyanUniversity of Michigan Medical School ann arbor
Arul M ChinnaiyanRogel Cancer Center, University of Michigan Medical School ann arbor
Arul M ChinnaiyanMichigan Center for Translational Pathology, University of Michigan Medical School ann arbor
Arul M ChinnaiyanRogel Cancer Center, University of Michigan Medical School ann arbor
Arul M ChinnaiyanUniversity of Michigan Medical School ann arbor
Arul M ChinnaiyanHoward Hughes Medical Institute, University of Michigan Medical School ann arbor
Kenneth AldapeCenter for Cancer Research, National Cancer Institute, National Institutes of Health
Kenneth AldapeCenter for Cancer Research, National Cancer Institute, National Institutes of Health
Stefan M PfisterHopp Children's Cancer Center (KiTZ) Heidelberg, German Cancer Research Center (DKFZ)
Stefan M PfisterHeidelberg University Hospital
Stefan M PfisterGerman Cancer Research Center (DKFZ)
Stefan M PfisterHopp Children's Cancer Center (KiTZ) Heidelberg, German Cancer Research Center (DKFZ)
Stefan M PfisterGerman Cancer Research Center (DKFZ)
Stefan M PfisterHeidelberg University Hospital
Mariella G Filbin
Mariella G FilbinBroad Institute of Harvard and MIT
Sriram VennetiUniversity of Michigan, 1150 W. Medical Center ann arbor
Sriram VennetiUniversity of Michigan ann arbor
Sriram VennetiUniversity of Michigan Medical School ann arbor
Sriram VennetiRogel Cancer Center, University of Michigan Medical School ann arbor
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