Skip to Main Content

Paper Details

Proteogenomics and Hi-C reveal transcriptional dysregulation in high hyperdiploid childhood acute lymphoblastic leukemia.
Nat Commun
55
2019
8,000 genes, ALL, CTCF, ETV6, RNA, RUNX1, TAD, acute lymphoblastic leukemia, childhood acute lymphoblastic leukemia, chromatin, chromosomes, cohesin, high, high hyperdiploid, hyperdiploid, hyperdiploid ALL, hyperdiploid childhood ALL, patient, primary, primary patient samples, topologically associating domain
Author NameAffiliation
Duncan T OdomCancer Research UK Cambridge Institute (CRUK-CI), University of Cambridge
Duncan T OdomGerman Cancer Research Center (DKFZ)
Duncan T OdomCancer Research UK Cambridge Institute (CRUK-CI), University of Cambridge
Duncan T OdomGerman Cancer Research Center (DKFZ)
Janne Lehti??Science for Life Laboratory and Karolinska Institute, Clinical Proteomics Mass Spectrometry
  • 1 - 5

Datasets

Release 1.2