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Paper Details

Characterization of the leukemogenic potential of distal cytoplasmic CSF3R truncation and missense mutations.
Leukemia
19
2017
CSF3R, CSF3R cytoplasmic domain, CSF3R cytoplasmic mutations, CSF3R truncation, Q793-Q823, STAT5, leukemia, patients
Alleles, Animals, Cell Line, Cell Transformation, Neoplastic, Humans, Leukemia, Myeloid, Acute, Mice, Mutation, Missense, Myeloproliferative Disorders, Phosphorylation, Protein Domains, Proteolysis, Receptors, Colony-Stimulating Factor, STAT5 Transcription Factor, Sequence Deletion
Author NameAffiliation
Haijiao ZhangOregon Health & Science University, Knight Cancer Institute
A Reister SchultzOregon Health & Science University, Knight Cancer Institute
Samuel B LutyOregon Health & Science University, Knight Cancer Institute
Angela RofeltyOregon Health & Science University, Knight Cancer Institute
Y SuOregon Health & Science University
Sophie MeansOregon Health & Science University, Knight Cancer Institute
Daniel BottomlyDepartment of Medical Informatics and Clinical Epidemiology, Oregon Health & Science University, Knight Cancer Institute
Beth WilmotDepartment of Medical Informatics and Clinical Epidemiology, Oregon Health & Science University, Knight Cancer Institute
Shannon K McWeeneyDepartment of Medical Informatics and Clinical Epidemiology, Oregon Health & Science University, Knight Cancer Institute
Jeffrey W TynerOregon Health & Science University, Knight Cancer Institute
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