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Paper Details

Myeloid lncRNA LOUP mediates opposing regulatory effects of RUNX1 and RUNX1-ETO in t(8;21) AML.
Blood
15
2021
-, AML, ETO, LOUP, LOUP locus, Myeloid lncRNA LOUP, PU, PU.1, RNA, RNAs, RUNX1, Runt-related transcription factor 1, acute myeloid leukemia, cell type, cell-type-specific RNAs, chromatin, gene loci, leukemia, lncRNA, long noncoding RNA, myeloid, myeloid gene, myeloid master regulator PU.1, myeloid-specific and polyadenylated lncRNA, t(8;, t(8;21, t(8;21) AML, upstream regulatory element of PU.1
Author NameAffiliation
Mahmoud A BassalHarvard Stem Cell Institute, Harvard University
Mahmoud A BassalCancer Science Institute of Singapore, National University of Singapore
Mahmoud A BassalHarvard Stem Cell Institute, Harvard University
Mahmoud A BassalCancer Science Institute of Singapore, National University of Singapore
Daniel G TenenBeth Israel Deaconess Medical Center, Harvard Medical School
Daniel G TenenBeth Israel Deaconess Medical Center, Harvard Medical School
Henry YangCancer Science Institute of Singapore, National University of Singapore
Henry YangCancer Science Institute of Singapore, National University of Singapore
Daniel G TenenHarvard Medical School Initiative for RNA Medicine, Harvard Medical School
Daniel G TenenStanford University School of Medicine
Daniel G TenenHarvard Medical School Initiative for RNA Medicine, Harvard Medical School
Daniel G TenenStanford University School of Medicine
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