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Paper Details

Aβ and tau prions feature in the neuropathogenesis of Down syndrome.
Proc Natl Acad Sci U S A
10
2022
A, AD, Alzheimer's disease, DS, Down syndrome, amyloid, chromosomal disorder, chromosome 21, dementia, double-, fAD, familial AD, humans, neurofibrillary tangles, people, prion disorder, sAD, sporadic AD, tau, tau prions
Author NameAffiliation
Carlo CondelloInstitute for Neurodegenerative Diseases, Weill Institute for Neurosciences, University of California san francisco
Carlo CondelloWeill Institute for Neurosciences, University of California san francisco
Alison M MaxwellDepartment of Pharmaceutical Chemistry, University of California san francisco
Erika CastilloInstitute for Neurodegenerative Diseases, Weill Institute for Neurosciences, University of California san francisco
Martin IngelssonUppsala University
Martin IngelssonUniversity of Toronto
Martin IngelssonKrembil Brain Institute, University Health Network
Lars LannfeltUppsala University
Thomas D BirdUniversity of Washington
Thomas D BirdUniversity of Washington
Christopher Dirk KeeneUniversity of Washington School of Medicine
William W SeeleyWeill Institute for Neurosciences, University of California san francisco
William W SeeleyUniversity of California san francisco
Daniel P PerlF. Edward Hebert School of Medicine, Uniformed Services University of the Health Sciences
Elizabeth HeadUniversity of California irvine
Stanley B PrusinerInstitute for Neurodegenerative Diseases, Weill Institute for Neurosciences, University of California san francisco
Stanley B PrusinerWeill Institute for Neurosciences, University of California san francisco
Stanley B PrusinerUniversity of California san francisco
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