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Paper Details

ATPase Inhibitory Factor-1 Disrupts Mitochondrial Ca Handling and Promotes Pathological Cardiac Hypertrophy through CaMKIIδ.
International Journal of Molecular Sciences
6
2021
ATP, ATP synthase, ATP-synthase, ATPase Inhibitory Factor-1, ATPase inhibitory factor-1, Ca2+, CaMKII, CaMKIIC, Cardiac Hypertrophy, E55A, HF, IF1, IF1 mutant, calcium, calcium-calmodulin kinase II, cardiomyocyte, cardiomyocyte hypertrophy, fetal-like gene program, heart failure, humans, hypertrophy, mice, mitochondrial dysfunction, pathological hypertrophy, primary cardiomyocytes, wild-type IF1
Author NameAffiliation
Rudolf A de BoerUniversity Medical Center Groningen, University of Groningen
Joan Heller BrownUniversity of California San Diego
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Datasets

Release 1.2