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Paper Details

SARS-CoV-2 infection enhances mitochondrial PTP complex activity to perturb cardiac energetics.
iScience
27
2022
ANT, ATP, ATP synthase, CCDC58, COVID-19, Ca, M-protein, NSP6, ORF10, ORF3A, ORF9C, PBMCs, SARS, SARS-, SARS-CoV, SARS-CoV-2, SARS-CoV-2 proteins, SPG-7, cardiomyocyte, coiled-coil domain containing protein 58, coronavirus disease 2019, cyclophilin D, cyclosporin A, human, mitochondrial PTP complex, respiratory disease
Author NameAffiliation
Gregory J AuneGreehey Children's Cancer Research Institute, University of Texas Health San Antonio
Luis Martinez-SobridoTexas Biomedical Research Institute
Muniswamy MadeshCenter for Precision Medicine, University of Texas Health San Antonio
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