Paper Details

Paper Title

Silencing of the Drosophila ortholog of SOX5 leads to abnormal neuronal development and behavioral impairment.

Paper Journal Title

Hum Mol Genet

Paper Citation Count

Paper Publication Year

2017

Bio Mention

AD, ALS, Drosophila, Drosophila ortholog of SOX5, LOAD, SH-SY5Y neuroblastoma cells, SOX5, SOX5 variants, Sox102F, WNT, WNT-related genes, amyotrophic lateral sclerosis, candidate gene, developmental delay, dysmorphic features, human, human SH-SY5Y neuroblastoma cells, impaired olfactory perception, intellectual disability, late-onset familial Alzheimer's disease, neurodegeneration in brain, neuronal subtypes, neurons, patients

Mesh Descriptor

Author Name	Affiliation
Brad A Chapman	Harvard T. H. Chan School of Public Health
Oliver Hofmann	Harvard T. H. Chan School of Public Health
Oliver Hofmann	Center for Cancer Research, University of Melbourne
Oliver Hofmann	Harvard T. H. Chan School of Public Health
Oliver Hofmann	Center for Cancer Research, University of Melbourne
Winston Hide	Harvard T. H. Chan School of Public Health
Winston Hide	Sheffield Institute for Translational Neuroscience, University of Sheffield
Winston Hide	Harvard T. H. Chan School of Public Health
Winston Hide	Sheffield Institute for Translational Neuroscience, University of Sheffield
Rudolph E Tanzi	Genetics and Aging Research Unit, Massachusetts General Hospital, Harvard Medical School, MassGeneral Institute for Neurodegenerative Diseases

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