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Paper Details

Alzheimer's disease-associated U1 snRNP splicing dysfunction causes neuronal hyperexcitability and cognitive impairment.
Nat Aging
14
2022
5xFAD amyloidosis, AD, Alzheimer's disease, GABA, GABA receptor, GABA receptor subunit, GABRA2, N, N40K, RNA, Tg, U1, U1 small nuclear ribonucleoprotein, U1 snRNP, U1-, U1-70K, amyloid, brain transcriptome, cognitive decline, cognitive impairment, full-, mice, neurodegeneration, neuronal degeneration, neuronal hyperexcitability, primary neurons
Author NameAffiliation
Bing BaiSt. Jude Children's Research Hospital
Bing BaiSt. Jude Children's Research Hospital
Bing BaiCenter for Precision Medicine, Nanjing Drum Tower Hospital, Nanjing University Medical School
Ji-Hoon ChoCenter for Proteomics and Metabolomics, St. Jude Children's Research Hospital
Elizabeth StewartSt. Jude Children's Research Hospital
Elizabeth StewartSt. Jude Children's Research Hospital
Elizabeth StewartSt. Jude Children's Research Hospital
Elizabeth StewartSt. Jude Children's Research Hospital
David A BennettRush University Medical Center
David A BennettRush University Medical Center
Taosheng ChenSt. Jude Children's Research Hospital
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