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Paper Details

New molecular mechanisms in cholangiocarcinoma: signals triggering interleukin-6 production in tumor cells and KRAS co-opted epigenetic mediators driving metabolic reprogramming.
J Exp Clin Cancer Res
7
2022
CAA cells, CCA, CCA cell, CCA cells, CCAs, CCl +, Cholangiocarcinogenesis, Cholangiocarcinoma, DEN, EGFR, G9a, Glucose, Huh28-KRAS cells, IL6, Jnk +, KRAS, KRAS CAA cells, Mutant KRAS, PHGDH, TAA, TAA-, [U-C]-D-glucose, amino acids, cancer, cholangiocarcinoma, epidermal growth factor receptor, genes, glucose, glycine, human, human CCA cells, iCCA, inflammation, interleukin-6, intrahepatic CCA, mice, mouse, mutant KRAS, phosphoglycerate, phosphoglycerate dehydrogenase, rat, rats, serine, thioacetamide, tumor, tumor cells, tumour, tumour cells

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