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Paper Details

Massively parallel sequencing of phyllodes tumours of the breast reveals actionable mutations, and TERT promoter hotspot mutations and TERT gene amplification as likely drivers of progression.
J Pathol
88
2016
-, 124 C>T, Borderline PTs, Breast fibroepithelial lesions, EGFR, MED12, MED12 exon 2, MED12 mutations, PTs, Phyllodes tumours, RB1, SETD2, TERT, TERT gene, TERT mRNA, TERT promoter, TP53, benign PTs, benign, borderline and malignant PTs, breast fibroepithelial lesions, cancer, cancer genes, fibroadenomas, malignant PTs, normal, phyllodes tumours of the breast
Author NameAffiliation
Salvatore PiscuoglioMemorial Sloan Kettering Cancer Center
Charlotte K Y NgMemorial Sloan Kettering Cancer Center
Melissa P MurrayMemorial Sloan Kettering Cancer Center
Kathleen A BurkeMemorial Sloan Kettering Cancer Center
Felipe C GeyerMemorial Sloan Kettering Cancer Center
Felipe C GeyerHospital Israelita Albert Einstein, Instituto Israelita de Ensino e Pesquisa
Luciano G MartelottoMemorial Sloan Kettering Cancer Center
Caterina Marchi??Memorial Sloan Kettering Cancer Center
Caterina Marchi??University of Turin
Raymond S LimMemorial Sloan Kettering Cancer Center
Larry NortonMemorial Sloan Kettering Cancer Center
Larry NortonMemorial Sloan Kettering Cancer Center
José BaselgaMemorial Sloan Kettering Cancer Center
José BaselgaMemorial Sloan Kettering Cancer Center
Marc LadanyiMemorial Sloan Kettering Cancer Center
Marc LadanyiMemorial Sloan Kettering Cancer Center
Britta WeigeltMemorial Sloan Kettering Cancer Center
Jorge S Reis-FilhoMemorial Sloan Kettering Cancer Center
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