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Paper Details

FRA1 contributes to MEK-ERK pathway-dependent PD-L1 upregulation by KRAS mutation in premalignant human bronchial epithelial cells.
Am J Transl Res
14
2020
AP-1 transcriptional factor superfamily, CCLE, Cancer Cell Line, EGFR mutation, ERK, FRA1, FRA1 mRNA, KRAS, KRAS mutated cells, KRAS mutation, KRAS mutationV12, MEK, NSCLC, Oncogenic KRAS mutations, PD-L1, Premalignant human bronchial epithelial cell (HBEC) lines, cancer, cancer types, human, lung cancer, non-small cell lung carcinoma, p53, pERK, premalignant, premalignant HBEC, premalignant human bronchial epithelial cells
Author NameAffiliation
Jane YanagawaJonsson Comprehensive Cancer Center Los Angeles
Jane YanagawaUniversity of California Los Angeles
Eileen FungJonsson Comprehensive Cancer Center Los Angeles
Eileen FungUniversity of California Los Angeles
Kostyantyn KrysanJonsson Comprehensive Cancer Center Los Angeles
Kostyantyn KrysanUniversity of California Los Angeles
John D MinnaHamon Center for Therapeutic Oncology Research The University of Texas Southwestern Medical Center Dallas
Steven M DubinettJonsson Comprehensive Cancer Center Los Angeles
Steven M DubinettUniversity of California Los Angeles
Steven M DubinettVeterans Affair Greater Los Angeles Healthcare System Los Angeles
Steven M DubinettJonsson Comprehensive Cancer Center Los Angeles
Steven M DubinettUniversity of California Los Angeles
Steven M DubinettVeterans Affair Greater Los Angeles Healthcare System Los Angeles
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