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Paper Details

FOXA1 mutations alter pioneering activity, differentiation and prostate cancer phenotypes.
Nature
135
2019
DNA, FOXA1, FOXA1 mutants, FOXA1 mutations, FOXA1-binding motifs, R219, R219 mutants, R219-mutant FOXA1, Wing2, Wing2 mutations, adenocarcinomas, cancer, chromatin, forkhead domain, genomic loci, human, metastatic tumours, mouse, mouse prostate organoid, non-canonical motif, open chromatin, prostate cancer, prostate cancers, transcription factor FOXA1, transposase, wild-type FOXA1
Author NameAffiliation
Robert N HooverMemorial Sloan Kettering Cancer Center
Robert N HooverMemorial Sloan Kettering Cancer Center
Zhigang ZhangMemorial Sloan Kettering Cancer Center
Zhigang ZhangMemorial Sloan Kettering Cancer Center
Yang Liu
Elai Davicioni
Andrea SbonerSandra and Edward Meyer Cancer Center
Andrea SbonerEnglander Institute for Precision Medicine of Weill Cornell Medicine and NewYork-Presbyterian Hospital
Andrea SbonerHRH Prince Alwaleed Bin Talal Bin Abdulaziz Alsaud Institute for Computational Biomedicine, Weill Cornell Medical College
Andrea SbonerSandra and Edward Meyer Cancer Center
Andrea SbonerHRH Prince Alwaleed Bin Talal Bin Abdulaziz Alsaud Institute for Computational Biomedicine, Weill Cornell Medical College
Andrea SbonerEnglander Institute for Precision Medicine of Weill Cornell Medicine and NewYork-Presbyterian Hospital
Christopher E BarbieriSandra and Edward Meyer Cancer Center
Christopher E BarbieriEnglander Institute for Precision Medicine of Weill Cornell Medicine and NewYork-Presbyterian Hospital
Christopher E Barbieri
Christopher E BarbieriSandra and Edward Meyer Cancer Center
Christopher E Barbieri
Christopher E BarbieriEnglander Institute for Precision Medicine of Weill Cornell Medicine and NewYork-Presbyterian Hospital
Rohit BoseUniversity of California san francisco
Christina S Leslie
Charles L SawyersMemorial Sloan Kettering Cancer Center
Charles L SawyersHoward Hughes Medical Institute
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