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Paper Details

Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.
PLoS Pathog
55
2014
C3, CD11c, IFN, IFNAR, Ifnar, Ifnar gene, Ifnar-/-, Ifnarf, MAVS, Mavs, Mavs-/-Ifnar-/- myeloid cells, RIG-I-like receptor adaptor gene, WNV, West Nile virus, factor B, inflammation, mice, myeloid cells, organ damage, sepsis, tissue injury, type I IFN, type I IFN receptor, type I IFN receptor (IFNAR) gene, type I interferon
Author NameAffiliation
Matthew J GormanWashington University School of Medicine
Mehul S SutharEmory University
Michael GaleUniversity of Washington School of Medicine
Michael S DiamondWashington University School of Medicine
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