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Paper Details

Pediatric T-ALL type-1 and type-2 relapses develop along distinct pathways of clonal evolution.
Leukemia
3
2022
BLM, BUB1B, IL7R, JAK, MDR1, MVP, NT5C2, PMS2, Patients, Pediatric T-ALL type, STAT, T, T-ALL, T-ALLs, TP53, cancer, cancer predisposition gene, immature thymic T-cell population, leukemia, major ancestral clone, minor ancestral clone, normal T-cell precursors, somatic, type, type-1 or type-2 T-ALL
Author NameAffiliation
Tobias RauschEMBL and Medical Faculty of the University of Heidelberg
Tobias Rausch
Yassen AssenovGerman Cancer Consortium (DKTK) and German Cancer Research Center (DKFZ)
Renate Kirschner-SchwabeCharite Universitatsmedizin Berlin
Renate Kirschner-Schwabeand German Cancer Research Center (DKFZ)
Cornelia EckertCharite Universitatsmedizin Berlin
Cornelia Eckertand German Cancer Research Center (DKFZ)
Stefan M PfisterUniversity of Heidelberg
Stefan M PfisterGerman Cancer Consortium (DKTK) and German Cancer Research Center (DKFZ)
Stefan M PfisterHopp Children's Cancer Center Heidelberg (KiTZ)
Stefan M PfisterUniversity of Heidelberg
Stefan M PfisterHopp Children's Cancer Center Heidelberg (KiTZ)
Stefan M PfisterGerman Cancer Consortium (DKTK) and German Cancer Research Center (DKFZ)
Jan O KorbelEMBL and Medical Faculty of the University of Heidelberg
Jan O Korbel
Jan O KorbelEMBL and Medical Faculty of the University of Heidelberg
Jan O Korbel
Andreas E KulozikUniversity of Heidelberg
Andreas E KulozikHopp Children's Cancer Center Heidelberg (KiTZ)
Andreas E KulozikEMBL and Medical Faculty of the University of Heidelberg
Andreas E KulozikGerman Cancer Consortium (DKTK) and German Cancer Research Center (DKFZ)
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