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Paper Details

Coronary Artery Disease Risk of Familial Hypercholesterolemia Genetic Variants Independent of Clinically Observed Longitudinal Cholesterol Exposure.
Circ Genom Precis Med
7
2022
Cholesterol, Coronary Artery Disease, FH, FH variant, FH variants, Familial Hypercholesterolemia, Familial hypercholesterolemia, LDL, LDL-C, Proprotein convertase subtilisin, acute myocardial infarction, cholesterol, coronary artery disease, kexin type 9, low-density lipoprotein cholesterol
Author NameAffiliation
Shoa L Clarke
Shoa L ClarkeStanford University School of Medicine
Catherine Tcheandjieu
Catherine TcheandjieuStanford University School of Medicine
Catherine Tcheandjieu
Catherine TcheandjieuStanford University School of Medicine
Julie A Lynch
Julie A LynchCollege of Nursing & Health Sciences, University of Massachusetts
Kyong-Mi ChangCorporal Michael J. Crescenz VA Medical Center
Kyong-Mi ChangUniversity of Pennsylvania Perelman School of Medicine
Joshua W KnowlesStanford University School of Medicine
Joshua W KnowlesCardiovascular Institute (J.W.K., Stanford University School of Medicine
Joshua W KnowlesDiabetes Research Center (J.W.K.), Stanford University School of Medicine
Christopher J O'DonnellVA Boston Healthcare System
Christopher J O'DonnellHarvard Medical School
Christopher J O'DonnellVA Boston Healthcare System
Christopher J O'DonnellHarvard Medical School
Philip S Tsao
Philip S TsaoCardiovascular Institute (J.W.K., Stanford University School of Medicine
Philip S TsaoStanford University School of Medicine
Philip S Tsao
Philip S TsaoStanford University School of Medicine
Philip S TsaoCardiovascular Institute (J.W.K., Stanford University School of Medicine
Daniel J RaderUniversity of Pennsylvania Perelman School of Medicine
John Michael GazianoVA Boston Healthcare System
John Michael GazianoVA Boston Healthcare System
Themistocles L Assimes
Themistocles L AssimesStanford University School of Medicine
Themistocles L AssimesCardiovascular Institute (J.W.K., Stanford University School of Medicine
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