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Paper Details

H3K4me3 remodeling induced acquired resistance through O-GlcNAc transferase.
Drug Resist Updat
1
2023
AMPK, CPG island, CPG island regions, DNA binding motifs, DTP, DTPP, DTPPs, GlcNAc, H3K4me3, H3K4me3 transcriptionally active sites, Histone H3, N-acetylglucosamine, O, O-GlcNAc, O-GlcNAc transferase, O-linked N-acetylglucosamine transferase, OGT, TET1, acetyl salicylic acid, cancer, cancer cell, cancer cells, chromatin, chronically treated cancer cells, drug tolerant persisters, drug tolerant proliferative persister, genes, lysine, patient, slow cycling, transcription factor binding motifs
Author NameAffiliation
Aik Choon TanUniversity of Colorado Denver
Aik Choon TanUniversity of Colorado Denver
Richard A SturmFrazer Institute, The University of Queensland
Richard A SturmFrazer Institute, The University of Queensland
Meenhard HerlynThe Wistar Institute
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